Out of Stock Free Shipping! A southern weed and feed that controls unwanted weeds and fertilizers the grass to protect it against heat and drought. Atrazine herbicide is used in crops such as corn and sugarcane. It provides good weed control for crops because it is a selective herbicide , meaning the crop plants can metabolize the chemical, but the weeds are killed. It also has lawn care uses, like for golf courses and residential lawns as a weed killer. It is great for dandelions and crab grass without killing your grass.
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Uses[ edit ] Atrazine is a herbicide that is used to stop pre- and post-emergence broadleaf and grassy weeds in crops such as sorghum , maize , sugarcane , lupins , pine , and eucalypt plantations, and triazine-tolerant canola. Like other triazine herbicides, atrazine functions by binding to the plastoquinone -binding protein in photosystem II , which animals lack.
Plant death results from starvation and oxidative damage caused by breakdown in the electron transport process. Oxidative damage is accelerated at high light intensity. Studies suggest that atrazine is an endocrine disruptor that can cause hormone imbalance. Atrazine biodegradation - atrazine chlorohydrolase pathway Atrazine remains in soil for a matter of months although in some soils can persist to at least 4 years  and can migrate from soil to groundwater ; once in groundwater, it degrades slowly.
It has been detected in groundwater at high levels in some regions of the U. The US Environmental Protection Agency expresses concern regarding contamination of surface waters lakes, rivers, and streams. The half-life of atrazine in soil ranges from 13 to days. The end product of this process is cyanuric acid , itself unstable with respect to ammonia and carbon dioxide.
The best characterized organisms that use this pathway are of Pseudomonas sp. Dealkylation of the amino groups gives 2-chlorohydroxyamino-1,3,5-triazine, the degradation of which is unknown. This path also occurs in Pseudomonas species, as well as a number of bacteria. Though the two alkyl moieties readily support growth of certain microorganisms, the atrazine ring is a poor energy source due to the oxidized state of ring carbon.
In fact, the most common pathway for atrazine degradation involves the intermediate, cyanuric acid, in which carbon is fully oxidized, thus the ring is primarily a nitrogen source for aerobic microorganisms. Atrazine may be catabolized as a carbon and nitrogen source in reducing environments, and some aerobic atrazine degraders have been shown to use the compound for growth under anoxia in the presence of nitrate as an electron acceptor,  a process referred to as a denitrification.
When atrazine is used as a nitrogen source for bacterial growth, degradation may be regulated by the presence of alternative sources of nitrogen. In pure cultures of atrazine-degrading bacteria, as well as active soil communities, atrazine ring nitrogen, but not carbon are assimilated into microbial biomass.
In Pseudomonas sp. ADP, the Atz genes are located noncontiguously on a plasmid with the genes for mercury catabolism. AtzA-C genes have also been found in a Gram-positive bacterium , but are chromosomally located.
In natural environments, some iron-bearing minerals are reduced by specific bacteria in the absence of oxygen, thus the abiotic transformation of herbicides by reduced minerals is viewed as "microbially induced".
The 1-hour inhalation LC50 is greater than 0. The 4-hour inhalation LC50 is 5. Atrazine is one of the most commonly used herbicides in the United States.
Studies of couples living on farms that use atrazine for weed control found an increase in the risk of preterm delivery, but these studies are difficult to interpret because most of the farmers were men who may have been exposed to several types of pesticides.
Little information is available regarding the risks to children, however "[m]aternal exposure to atrazine in drinking water has been associated with low fetal weight and heart, urinary, and limb defects in humans". In people, risks for preterm delivery and intrauterine growth retardation have been associated with exposure.
Atrazine exposure has been shown to result in delays or changes in pubertal development in female rats; conflicting results have been observed in males. Male rats exposed via milk from orally exposed mothers exhibited higher levels of prostate inflammation as adults; immune effects have also been seen in male rats exposed in utero or while nursing. The study tracked 57, licensed pesticide applicators over 13 years. Reproductive effects in rats and rabbits were only seen at doses that were toxic to the mother.
The authors concluded that the quality of most studies was poor and without good quality data, the results were difficult to assess, though it was noted that no single category of negative pregnancy outcome was found consistently across studies. The authors concluded that a causal link between atrazine and adverse pregnancy outcomes was not warranted due to the poor quality of the data and the lack of robust findings across studies. Syngenta was not involved in the design, collection, management, analysis, or interpretation of the data and did not participate in the preparation of the manuscript.
Its principal finding was that susceptibility of wood frog tadpoles to infection by E. Tissue malformation may have been induced by ectopic programmed cell death , although a mechanism was not identified. The authors concluded, "Statements by Hayes and Syngenta suggest that their scientific differences have developed a personal aspect that casts doubt on their scientific objectivity".
The company denied all wrongdoing.
Helena Atrazine 90-DG